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Anders Åhlin

Universitetslektor/överläkare

Besöksadress : Sjukhusbacken 10 118 83 Stockholm
Postadress : Institutionen för klinisk forskning och utbildning, Södersjukhuset (KI SÖS), S1, Institutionen för klinisk forskning och utbildning, Södersjukhuset (KI SÖS), S1, Sjukhusbacken 10 118 83 Stockholm
Leveransadress : Sjukhusbacken 10 118 83 Stockholm

Publikationer

[Chronic granulomatous disease--not only the lack of oxygen radicals]
Palmblad J, Gyllenhammar H, Bratt J, Ahlin A
Lakartidningen 2005;102(14):1036-8

Marked variability in clinical presentation and outcome of patients with C1q immunodeficiency
Van Schaarenburg Ra, Schejbel L, Truedsson L, Topaloglu R, Al-mayouf Sm, Riordan A, et al
Journal of autoimmunity 2015;62():39-44

Mutations in the X-linked and autosomal recessive forms of chronic granulomatous disease
Roos D, Deboer M, Kuribayashi F, Meischl C, Weening Rs, Segal Aw, et al
BLOOD 1996;87(5):1663-81

Chronic granulomatous disease: The European experience
Van Den Bjm, Van Koppen E, Ahlin A, Belohradsky Bh, Bernatowska E, Bernatowska E, et al
CLINICAL AND EXPERIMENTAL IMMUNOLOGY 2008;:206-206

Chronic granutomatous disease caused by mutations other than the common GT deletion in NCF1, the gene encoding the p47(phox) component of the phagocyte NADPH oxidase
Roos D, De Boer M, Koker My, Dekker J, Singh-gupta V, Ahlin A, et al
HUMAN MUTATION 2006;27(12):1218-29

Abberant cytosolic calcium ion mobilization in chronic granulomatous disease neutrophils
Palmblad J, Hansson A, Heimburger M, Ahlin A
INFLAMMATION 2004;28(3):133-8

Prevalence, genetics and clinical presentation of chronic granulomatous disease in Sweden
Ahlin A, Deboer M, Roos D, Leusen J, Smith Cie, Sundin U, et al
ACTA PAEDIATRICA 1995;84(12):1386-94

NEUTROPHIL MEMBRANE-POTENTIAL CHANGES AND HOMOTYPIC AGGREGATION KINETICS ARE PH-DEPENDENT - STUDIES OF CHRONIC GRANULOMATOUS-DISEASE
Ahlin A, Gyllenhammar H, Ringertz B, Palmblad J
JOURNAL OF LABORATORY AND CLINICAL MEDICINE 1995;125(3):392-401

Disturbed interaction of p21-rac with mutated p67-phox causes chronic granulomatous disease
Leusen Jhw, Deklein A, Hilarius Pm, Ahlin A, Palmblad J, Smith Cie, et al
JOURNAL OF EXPERIMENTAL MEDICINE 1996;184(4):1243-9

Dose-dependent enhancements by interferon-gamma on functional responses of neutrophils from chronic granulomatous disease patients
Ahlin A, Elinder G, Palmblad J
BLOOD 1997;89(9):3396-401

Involvement of caspases in neutrophil apoptosis: Regulation by reactive oxygen species
Fadeel B, Ahlin A, Henter Ji, Orrenius S, Hampton Mb
BLOOD 1998;92(12):4808-18

Gamma interferon treatment of patients with chronic granulomatous disease is associated with augmented production of nitric oxide by polymorphonuclear neutrophils
Ahlin A, Larfars G, Elinder G, Palmblad J, Gyllenhammar H
CLINICAL AND DIAGNOSTIC LABORATORY IMMUNOLOGY 1999;6(3):420-4

Four novel mutations in the gene encoding gp91-phox of human NADPH oxidase: consequences for oxidase assembly
Leusen Jhw, Meischl C, Eppink Mhm, Hilarius Pm, De Boer M, Weening Rs, et al
BLOOD 2000;95(2):666-73

A new exon created by intronic insertion of a rearranged LINE-1 element as the cause of chronic granulomatous disease
Meischl C, De Boer M, Ahlin A, Roos D
EUROPEAN JOURNAL OF HUMAN GENETICS 2000;8(9):697-703

Efficacy and safety of two different rG-CSF preparations in the treatment of patients with severe chronic neutropenia.
Carlsson G, Ahlin A, Elinder G, Dahllof G, Henter Ji, Palmblad J
BLOOD 2003;102(11):165B-166B

Bone marrow transplantation in chronic granulomatous disease - A comparison with conventional treatment and a presentation of two new cases
Ahlin A, Ringden O, Winiarski J
PEDIATRIC RESEARCH 2003;53(4):293A-293A

Efficacy and safety of two different rG-CSF preparations in the treatment of patients with severe congenital neutropenia
Carlsson G, Ahlin A, Dahllof G, Elinder G, Henter Ji, Palmblad J
BRITISH JOURNAL OF HAEMATOLOGY 2004;126(1):127-32

Swedish guidelines for the assessment, diagnosis and management of 6 primary immunodeficiency states: CVID, IgG subclass deficiency, IgA deficiency, XLA, SCID and CGD
Brodszki Nb, Matsols H, Fasth A, Friman V, Lofdahl K, Oskarsdottir S, et al
CLINICAL AND EXPERIMENTAL IMMUNOLOGY 2008;:140-141

Involvement of a functional NADPH oxidase in neutrophils and macrophages during programmed cell clearance: implications for chronic granulomatous disease
Sanmun D, Witasp E, Jitkaew S, Tyurina Yy, Kagan Ve, Ahlin A, et al
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY 2009;297(3):C621-31

Molecular basis of hereditary C1q deficiency-revisited: identification of several novel disease-causing mutations
Schejbel L, Skattum L, Hagelberg S, Ahlin A, Schiller B, Berg S, et al
GENES AND IMMUNITY 2011;12(8):626-34

Allogeneic Haematopoietic Stem Cell Transplantation in the Treatment for Human C1q Deficiency - The Karolinska Experience
Olsson R, Hagelberg S, Schiller B, Ringden O, Truedsson L, Ahlin A
BONE MARROW TRANSPLANTATION 2015;:S489-S489

Chronic granulomatous disease - conventional treatment vs. hematopoietic stem cell transplantation: an update
Åhlin A, Fasth A
Current opinion in hematology 2015;22(1):41-5

Chronic granulomatous disease-haematopoietic stem cell transplantation versus conventional treatment
Åhlin A, Fugeläng J, De Boer M, Ringden O, Fasth A, Winiarski J
Acta paediatrica (Oslo, Norway : 1992) 2013;102(11):1087-94

Chronic granulomatous disease: the European experience
Van Den Berg Jm, Van Koppen E, Ahlin A, Belohradsky Bh, Bernatowska E, Corbeel L, et al
PloS one 2009;4(4):e5234-

Allogeneic haematopoietic stem cell transplantation in the treatment of human C1q deficiency
Olsson R, Hagelberg S, Schiller B, Ringden O, Ahlin A
MOLECULAR IMMUNOLOGY 2015;67(1):168-168

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