CCK Extra Seminar: PhD Michael Resnick
"The influence of p53 and mutations on immune/inflammatory responses"
Michael Resnick, National Institute of Environmental Health Sciences, NIH, USA
Host: Klas Wiman
Coincident resection at both ends of random, γ-induced double-strand breaks requires MRX (MRN), Sae2 (Ctp1), and Mre11-nuclease.
PLoS Genet. 2013 Mar;9(3):e1003420
Clustered mutations in yeast and in human cancers can arise from damaged long single-strand DNA regions.
Mol. Cell 2012 May;46(4):424-35
Differential effects of poly(ADP-ribose) polymerase inhibition on DNA break repair in human cells are revealed with Epstein-Barr virus.
Proc. Natl. Acad. Sci. U.S.A. 2012 Apr;109(17):6590-5
The Toll-like receptor gene family is integrated into human DNA damage and p53 networks.
PLoS Genet. 2011 Mar;7(3):e1001360
Cohesin Is limiting for the suppression of DNA damage-induced recombination between homologous chromosomes.
PLoS Genet. 2010 Jul;6(7):e1001006