Lectures and seminars

CCK Extra Seminar: PhD Michael Resnick

2014-06-2412:30 Radiumhemmets föreläsningssal P1:01, Karolinska University HospitalKarolinska University Hospital, Solna

"The influence of p53 and mutations on immune/inflammatory responses"


Michael Resnick, National Institute of Environmental Health Sciences, NIH, USA
Host: Klas Wiman

Selected publications

Coincident resection at both ends of random, γ-induced double-strand breaks requires MRX (MRN), Sae2 (Ctp1), and Mre11-nuclease.
Westmoreland J, Resnick M
PLoS Genet. 2013 Mar;9(3):e1003420

Clustered mutations in yeast and in human cancers can arise from damaged long single-strand DNA regions.
Roberts S, Sterling J, Thompson C, Harris S, Mav D, Shah R, et al
Mol. Cell 2012 May;46(4):424-35

Differential effects of poly(ADP-ribose) polymerase inhibition on DNA break repair in human cells are revealed with Epstein-Barr virus.
Ma W, Halweg C, Menendez D, Resnick M
Proc. Natl. Acad. Sci. U.S.A. 2012 Apr;109(17):6590-5

The Toll-like receptor gene family is integrated into human DNA damage and p53 networks.
Menendez D, Shatz M, Azzam K, Garantziotis S, Fessler M, Resnick M
PLoS Genet. 2011 Mar;7(3):e1001360

Cohesin Is limiting for the suppression of DNA damage-induced recombination between homologous chromosomes.
Covo S, Westmoreland J, Gordenin D, Resnick M
PLoS Genet. 2010 Jul;6(7):e1001006


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